Eficácia clínica das enzimas sistêmicas Clinical Efficacy of Systemic Enzyme Support

Clinical observations and literature review both affirm the conviction that systemic enzyme support with Wobenzym N or Wobenzym PS is an essential component for successful management of inflammation disorders and other conditions with immune system dysregulation due to its high degree of clinical efficacy. In addition to improving clinical outcomes in conditions with overt inflammation, such as rheumatoid arthritis, thrombophlebitis, pyelonephritis, prostatitis and psoriasis, systemic enzyme support is also affective in conditions with covert inflammation such as osteoarthritis, angina, atherosclerosis, myocardial infarctions, and diabetes to name a few. The adjuvant properties of systemic enzyme support have also been observed and documented for a number of cases including adnexitis, arthritis, papillomas and various forms of cancer. This article will familiarize clinicians with the therapeutic benefits of systemic enzyme support and review pertinent findings related to this treatment modality. Enzymes are biological molecules that increase the rate of chemical reactions. In the human body, thousands of chemical reactions occur during the course of normal metabolic processes. These reactions require significant energy in order to take place. Enzymes act as catalyst to lower the energy needed for the reaction to move forward. As such, enzymes maintain optimal function of the various systems in the body and support overall good health and optimal quality of life. The immune system is very dependent on proper enzyme function in regards to regulating inflammation as well as protecting cells from damage. Cytokine activity, and the clearance of excessive inflammatory cytokines, is regulated by proteases, enzymes which degrade proteins. The clearance of tissue proteins and peptides damaged by inflammation is also mediated by proteases. Proteases significantly reduce concentrations of advance glycation endproducts (AGEs) and protect cells by decreasing their receptor (RAGEs) activation. Proteases also down-regulate adhesion molecule activity in inflamed, as well as malignant cells.